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rat il 6 cat  (Elabscience Biotechnology)


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    Elabscience Biotechnology rat il 6 cat
    Effect of pirfenidone and quercetin on the level of pulmonary inflammatory drive in the different experimental groups: (A) Represents the quantification of TNF‐α in lung tissue. (B) Illustrates the concentrations of IL‐1β and (C) <t>shows</t> <t>IL‐6</t> expression. All measured in pg/g protein. Bars represent the average of 5 measurements ( n = 5) and error bars represent standard error (SEM). Significance is denoted by symbols above the bars: * p < 0.05, ** p < 0.01, *** p < 0.001 compared to the control group; + p < 0.05, ++ p < 0.01, +++ p < 0.001 compared to the BLM group; # p < 0.05, ## p < 0.01, ### p < 0.001 compared to the Pirf group, $ p < 0.05, $$ p < 0.01, $$$ p < 0.001 compared to the Quer group.
    Rat Il 6 Cat, supplied by Elabscience Biotechnology, used in various techniques. Bioz Stars score: 96/100, based on 401 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/e+el+r0015+cat/pmc12965903-98-31-36?v=Elabscience+Biotechnology
    Average 96 stars, based on 401 article reviews
    rat il 6 cat - by Bioz Stars, 2026-07
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    Images

    1) Product Images from "Targeting Inflammatory and Oxidative Pathways in Idiopathic Pulmonary Fibrosis: Synergistic Effects of Quercetin and Pirfenidone in Modulating Nrf2/ PPAR ‐γ and TLR ‐4/ NF ‐ κB Pathways"

    Article Title: Targeting Inflammatory and Oxidative Pathways in Idiopathic Pulmonary Fibrosis: Synergistic Effects of Quercetin and Pirfenidone in Modulating Nrf2/ PPAR ‐γ and TLR ‐4/ NF ‐ κB Pathways

    Journal: Pharmacology Research & Perspectives

    doi: 10.1002/prp2.70229

    Effect of pirfenidone and quercetin on the level of pulmonary inflammatory drive in the different experimental groups: (A) Represents the quantification of TNF‐α in lung tissue. (B) Illustrates the concentrations of IL‐1β and (C) shows IL‐6 expression. All measured in pg/g protein. Bars represent the average of 5 measurements ( n = 5) and error bars represent standard error (SEM). Significance is denoted by symbols above the bars: * p < 0.05, ** p < 0.01, *** p < 0.001 compared to the control group; + p < 0.05, ++ p < 0.01, +++ p < 0.001 compared to the BLM group; # p < 0.05, ## p < 0.01, ### p < 0.001 compared to the Pirf group, $ p < 0.05, $$ p < 0.01, $$$ p < 0.001 compared to the Quer group.
    Figure Legend Snippet: Effect of pirfenidone and quercetin on the level of pulmonary inflammatory drive in the different experimental groups: (A) Represents the quantification of TNF‐α in lung tissue. (B) Illustrates the concentrations of IL‐1β and (C) shows IL‐6 expression. All measured in pg/g protein. Bars represent the average of 5 measurements ( n = 5) and error bars represent standard error (SEM). Significance is denoted by symbols above the bars: * p < 0.05, ** p < 0.01, *** p < 0.001 compared to the control group; + p < 0.05, ++ p < 0.01, +++ p < 0.001 compared to the BLM group; # p < 0.05, ## p < 0.01, ### p < 0.001 compared to the Pirf group, $ p < 0.05, $$ p < 0.01, $$$ p < 0.001 compared to the Quer group.

    Techniques Used: Expressing, Control



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    Effect of pirfenidone and quercetin on the level of pulmonary inflammatory drive in the different experimental groups: (A) Represents the quantification of TNF‐α in lung tissue. (B) Illustrates the concentrations of IL‐1β and (C) <t>shows</t> <t>IL‐6</t> expression. All measured in pg/g protein. Bars represent the average of 5 measurements ( n = 5) and error bars represent standard error (SEM). Significance is denoted by symbols above the bars: * p < 0.05, ** p < 0.01, *** p < 0.001 compared to the control group; + p < 0.05, ++ p < 0.01, +++ p < 0.001 compared to the BLM group; # p < 0.05, ## p < 0.01, ### p < 0.001 compared to the Pirf group, $ p < 0.05, $$ p < 0.01, $$$ p < 0.001 compared to the Quer group.
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    Effect of pirfenidone and quercetin on the level of pulmonary inflammatory drive in the different experimental groups: (A) Represents the quantification of TNF‐α in lung tissue. (B) Illustrates the concentrations of IL‐1β and (C) <t>shows</t> <t>IL‐6</t> expression. All measured in pg/g protein. Bars represent the average of 5 measurements ( n = 5) and error bars represent standard error (SEM). Significance is denoted by symbols above the bars: * p < 0.05, ** p < 0.01, *** p < 0.001 compared to the control group; + p < 0.05, ++ p < 0.01, +++ p < 0.001 compared to the BLM group; # p < 0.05, ## p < 0.01, ### p < 0.001 compared to the Pirf group, $ p < 0.05, $$ p < 0.01, $$$ p < 0.001 compared to the Quer group.
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    Effect of pirfenidone and quercetin on the level of pulmonary inflammatory drive in the different experimental groups: (A) Represents the quantification of TNF‐α in lung tissue. (B) Illustrates the concentrations of IL‐1β and (C) <t>shows</t> <t>IL‐6</t> expression. All measured in pg/g protein. Bars represent the average of 5 measurements ( n = 5) and error bars represent standard error (SEM). Significance is denoted by symbols above the bars: * p < 0.05, ** p < 0.01, *** p < 0.001 compared to the control group; + p < 0.05, ++ p < 0.01, +++ p < 0.001 compared to the BLM group; # p < 0.05, ## p < 0.01, ### p < 0.001 compared to the Pirf group, $ p < 0.05, $$ p < 0.01, $$$ p < 0.001 compared to the Quer group.
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    Effect of pirfenidone and quercetin on the level of pulmonary inflammatory drive in the different experimental groups: (A) Represents the quantification of TNF‐α in lung tissue. (B) Illustrates the concentrations of IL‐1β and (C) <t>shows</t> <t>IL‐6</t> expression. All measured in pg/g protein. Bars represent the average of 5 measurements ( n = 5) and error bars represent standard error (SEM). Significance is denoted by symbols above the bars: * p < 0.05, ** p < 0.01, *** p < 0.001 compared to the control group; + p < 0.05, ++ p < 0.01, +++ p < 0.001 compared to the BLM group; # p < 0.05, ## p < 0.01, ### p < 0.001 compared to the Pirf group, $ p < 0.05, $$ p < 0.01, $$$ p < 0.001 compared to the Quer group.
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    Effect of GdCl 3 treatment on systemic and intestinal inflammatory responses following CLP-induced sepsis. Levels of (A) TNF-α in sera, (B) TNF-α in intestinal tissue, (C) IL-1β in sera, (D) IL-1β in intestinal tissue (E) <t>IL-6</t> in serum and (F) IL-6 in intestinal tissue of CLP-treated rats with or without GdCl 3 pretreatment. Data were measured at 6, 12 and 24 h after CLP operation and are presented as the mean ± SD (n=6). * P<0.05 vs. sham group at the same time point; # P<0.05 vs. CLP group at the same time point; ns, not significant vs. sham group at the same time point; NS, not significant vs. CLP group at 24 h. GdCl 3 , gadolinium chloride; CLP, cecal ligation and puncture; TNF-α, tumor necrosis factor-α; IL-1β, interleukin-1β; IL-6, <t>interleukin-6.</t>
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    Effect of GdCl 3 treatment on systemic and intestinal inflammatory responses following CLP-induced sepsis. Levels of (A) TNF-α in sera, (B) TNF-α in intestinal tissue, (C) IL-1β in sera, (D) IL-1β in intestinal tissue (E) <t>IL-6</t> in serum and (F) IL-6 in intestinal tissue of CLP-treated rats with or without GdCl 3 pretreatment. Data were measured at 6, 12 and 24 h after CLP operation and are presented as the mean ± SD (n=6). * P<0.05 vs. sham group at the same time point; # P<0.05 vs. CLP group at the same time point; ns, not significant vs. sham group at the same time point; NS, not significant vs. CLP group at 24 h. GdCl 3 , gadolinium chloride; CLP, cecal ligation and puncture; TNF-α, tumor necrosis factor-α; IL-1β, interleukin-1β; IL-6, <t>interleukin-6.</t>
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    Image Search Results


    Effect of pirfenidone and quercetin on the level of pulmonary inflammatory drive in the different experimental groups: (A) Represents the quantification of TNF‐α in lung tissue. (B) Illustrates the concentrations of IL‐1β and (C) shows IL‐6 expression. All measured in pg/g protein. Bars represent the average of 5 measurements ( n = 5) and error bars represent standard error (SEM). Significance is denoted by symbols above the bars: * p < 0.05, ** p < 0.01, *** p < 0.001 compared to the control group; + p < 0.05, ++ p < 0.01, +++ p < 0.001 compared to the BLM group; # p < 0.05, ## p < 0.01, ### p < 0.001 compared to the Pirf group, $ p < 0.05, $$ p < 0.01, $$$ p < 0.001 compared to the Quer group.

    Journal: Pharmacology Research & Perspectives

    Article Title: Targeting Inflammatory and Oxidative Pathways in Idiopathic Pulmonary Fibrosis: Synergistic Effects of Quercetin and Pirfenidone in Modulating Nrf2/ PPAR ‐γ and TLR ‐4/ NF ‐ κB Pathways

    doi: 10.1002/prp2.70229

    Figure Lengend Snippet: Effect of pirfenidone and quercetin on the level of pulmonary inflammatory drive in the different experimental groups: (A) Represents the quantification of TNF‐α in lung tissue. (B) Illustrates the concentrations of IL‐1β and (C) shows IL‐6 expression. All measured in pg/g protein. Bars represent the average of 5 measurements ( n = 5) and error bars represent standard error (SEM). Significance is denoted by symbols above the bars: * p < 0.05, ** p < 0.01, *** p < 0.001 compared to the control group; + p < 0.05, ++ p < 0.01, +++ p < 0.001 compared to the BLM group; # p < 0.05, ## p < 0.01, ### p < 0.001 compared to the Pirf group, $ p < 0.05, $$ p < 0.01, $$$ p < 0.001 compared to the Quer group.

    Article Snippet: The assay was performed using ELISA kits for rat TNF‐α, cat. no.: ELK1396 (ELK Biotechnology, Wuhan, Hubei, China), rat IL‐1β ELISA Kit, cat. no.: ER1094 (Fine Biotech, Wuhan, Hubei, China), and rat IL‐6, cat. no.: E‐EL‐R0015 (Elabscience Biotechnology, USA).

    Techniques: Expressing, Control

    Effect of GdCl 3 treatment on systemic and intestinal inflammatory responses following CLP-induced sepsis. Levels of (A) TNF-α in sera, (B) TNF-α in intestinal tissue, (C) IL-1β in sera, (D) IL-1β in intestinal tissue (E) IL-6 in serum and (F) IL-6 in intestinal tissue of CLP-treated rats with or without GdCl 3 pretreatment. Data were measured at 6, 12 and 24 h after CLP operation and are presented as the mean ± SD (n=6). * P<0.05 vs. sham group at the same time point; # P<0.05 vs. CLP group at the same time point; ns, not significant vs. sham group at the same time point; NS, not significant vs. CLP group at 24 h. GdCl 3 , gadolinium chloride; CLP, cecal ligation and puncture; TNF-α, tumor necrosis factor-α; IL-1β, interleukin-1β; IL-6, interleukin-6.

    Journal: Experimental and Therapeutic Medicine

    Article Title: Gadolinium chloride pre-treatment reduces the inflammatory response and preserves intestinal barrier function in a rat model of sepsis

    doi: 10.3892/etm.2021.10577

    Figure Lengend Snippet: Effect of GdCl 3 treatment on systemic and intestinal inflammatory responses following CLP-induced sepsis. Levels of (A) TNF-α in sera, (B) TNF-α in intestinal tissue, (C) IL-1β in sera, (D) IL-1β in intestinal tissue (E) IL-6 in serum and (F) IL-6 in intestinal tissue of CLP-treated rats with or without GdCl 3 pretreatment. Data were measured at 6, 12 and 24 h after CLP operation and are presented as the mean ± SD (n=6). * P<0.05 vs. sham group at the same time point; # P<0.05 vs. CLP group at the same time point; ns, not significant vs. sham group at the same time point; NS, not significant vs. CLP group at 24 h. GdCl 3 , gadolinium chloride; CLP, cecal ligation and puncture; TNF-α, tumor necrosis factor-α; IL-1β, interleukin-1β; IL-6, interleukin-6.

    Article Snippet: ELISA kits from Elabscience Biotechnology Inc. were used to assess the concentrations of TNF-α (cat. no. E-EL-R0019), IL-6 (cat. no. E-EL-R0015) and IL-1β (cat. no. E-EL-R0012) in rat serum or supernatant from intestinal tissue homogenization.

    Techniques: Ligation